Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Aug 26;11:2081. doi: 10.3389/fimmu.2020.02081

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2020 Halbgebauer, Karasu, Braun, Palmer, Braumüller, Schultze, Schäfer, Bückle, Eigner, Wachter, Radermacher, Resuello, Tuplano, Nilsson Ekdahl, Nilsson, Armacki, Kleger, Seufferlein, Kalbitz, Gebhard, Lambris, van Griensven and Huber-Lang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

PMC Copyright notice

Non-receptor tyrosine-protein kinase 1 (TNK1) expression in murine kidneys after HS. TNK1 expression in kidneys of control (Ctrl) mice and animals after hemorrhagic shock (HS) was assessed by immunohistochemical staining and semiquantitative analysis. While there was only a modest increase in TNK1 expression (stained in red; nuclei were counterstained with hematoxylin) in the renal cortex (A,B), expression was significantly higher in collecting ducts of animals after HS (A,C) compared to controls. Quantitative real-time PCR revealed a significant increase in TNK1 mRNA in whole tissue lysates (D). *p < 0.05; n = 4 Ctrl, and n = 4 HS for (B,C); n = 4 Ctrl, n = 7 HS for (D).