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. 2020 Aug 13;7(9):1681–1691. doi: 10.1002/acn3.51155

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© 2020 The Authors. Annals of Clinical and Translational Neurology published by Wiley Periodicals LLC on behalf of American Neurological Association

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Representative FDG and PiB PET patterns in patients with AD and NCs. The left two rows show SPM T‐maps (P < 0.001) rendered on the ch2bet template brain. The right two rows display axial images of FDG PET and PiB PET, respectively. Hypometabolism in inferior parietal lobule, precuneus/posterior cingulate gyrus, bilateral dorsolateral frontal lobe, and medial prefrontal cortex on FDG PET and amyloid positivity on PiB PET are seen in a patient with AD. Normal glucose metabolism on FDG PET and amyloid negativity on PiB PET from a healthy individual are also presented. Abbreviations: FDG, fluorodeoxyglucose; SPM, statistical parametric mapping; PiB, Pittsburgh Compound‐B; PET, positron emission tomography; AD, Alzheimer’s disease; NC, normal control.