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. 2020 Aug 27;8:833. doi: 10.3389/fcell.2020.00833

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Copyright © 2020 Deng, Chen, Zhang, Li, He and Tan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Induction of mitochondrial dynamics disorder abolishes AMPK2α overexpression-mediated cardioprotection. (A,B) TUNEL staining was applied to observe cell apoptosis. Cardiomyocytes transfected with AMPKα2 adenovirus were treated with FCCP to induce mitochondrial dynamics disorder. Then, the number of TUNEL-positive cells was recorded. (C) LDH release assay was used to measure cell viability. (D–F) The cardiomyocytes contractile properties in the context of H/R injury. The data represent the mean ± SEM. P < 0.05.