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. 2020 Sep 9;11(9):728. doi: 10.1038/s41419-020-02926-8

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© The Author(s) 2020, corrected publication 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Western blotting of the Akt/mTOR axis in four groups: sh-NC, sh-PCAT6#1, sh-PCAT6#1 + pcDNA3.1, and sh-PCAT6#1 + pcDNA3.1/VEGFR2 group. b, c EdU (scale bar: 100 μm) and colony-formation assay revealed TNBC cell proliferation ability in four groups. d, e Transwell (scale bar: 200 μm) and wound-healing assay (scale bar: 100 μm) demonstrated TNBC cell invasion ability in four groups. f, g Western blot and immunofluorescence staining assay revealed TNBC cell EMT ability in four groups. h Tube-formation assay illustrated TNBC angiogenesis ability in four groups (scale bar: 200 μm). **P < 0.01. n.s. no significance.