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. 2020 Sep 10;33(12):789–815. doi: 10.1089/ars.2019.7800

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© Lydie Plecitá-Hlavatá et al. 2020; Published by Mary Ann Liebert, Inc.

This Open Access article is distributed under the terms of the Creative Commons License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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FIG. 9. — Effects of suppressors of electron leak at specific sites. (A) Effect of S1QEL—Mitochondrial matrix superoxide release J_m rates were normalized to those without S1QEL at varying glucose concentrations in the absence and presence of rotenone as indicated. (B) Antioxidant capacity of S3QEL—J_m rates were normalized to those without S3QEL in the absence or presence of rotenone. (C) Simultaneous effects of S1QEL plus S3QEL at 11 mM glucose. J_m rates were normalized to those without both agents. (D) Antioxidant effect of S3QEL towards Antimycin-induced superoxide production. S1QEL and S3QEL, 10 μM; rotenone, 20 μM. ANOVA (n = 4–12): ^##p < 0.01; ^###p < 0.001; when normalized to 100%. **p < 0.01; ***p < 0.001; when compared among samples. S3QEL, suppressor of complex 3 site Q electron leak.