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. 2020 Aug 28;11:2024. doi: 10.3389/fimmu.2020.02024

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Copyright © 2020 Yin, Fan, Xu, Wu, Li, Zhou, Liao, Duan, Wang, Geng and Jin.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The stimulatory roles of EVs in lung cancer immunity. (a) Heat stressed, (b) Rab27a overexpressed, and (c) CD40L overexpressed lung cancer cell–derived exosomes (TEXs) can activate DC maturation and arouse specific antitumor immunity. (d) Activated dendritic cell–derived exosomes (DEXs) can present antigens to T-cells directly or by transferring pMHC to other immature dendritic cells and amplifing this antigen-presentation effect. TCR, T-cell receptor. Figure created with BioRender.com.