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. Author manuscript; available in PMC: 2021 Jul 21.
Published in final edited form as: J Neurosurg. 2020 Mar 6;134(3):902–908. doi: 10.3171/2019.12.JNS193023

Table 1.

Baseline characteristics of the sample.

Variable Patients = 74
Aneurysms = 96
Age (in years, mean ± SD) 64.7 ± 12.4
Women (%) 60 (81)
Smoking (%) 40 (54.1)
Hypertension (%) 41 (55.4)
Diabetes (%) 12 (16.2)
Hyperlipidemia (%) 32 (43.2)
Family history of intracranial aneurysms (%) 7 (9.5)
History of previous SAH (%) 4 (5.4)
Aspirin* 39 (52.7)
Size (in mm, mean, range) 8.1 (3-27)
- ≥7 mm (%) 48 (50.0)
- <7 mm (%) 48 (50.0)
Location
- Internal carotid artery (%) 25 (26.0)
- Anterior communicating artery (%) 16 (16.7)
- Anterior cerebral artery (%) 5 (5.2)
- Middle cerebral artery (%) 26 (27.1)
- Posterior communicating artery (%) 5 (5.2)
- Basilar artery (%) 15 (15.6)
- Superior cerebellar (%) 3 (3.1)
- Vertebral artery (%) 1 (1.0)
Aneurysmal wall enhancement
- Positive 59 (61.5%)
- Negative 37 (38.5%)

mm: millimeters; SD: standard deviation

*

Refers to daily intake of ≥81 mg of aspirin during the last 6 months.

Defined as positive if aneurysm-to-pituitary stalk contrast ratio (CRstalk) > 0.60.