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. Author manuscript; available in PMC: 2021 Oct 1.
Published in final edited form as: Neurotox Res. 2020 Jun 30;38(3):808–823. doi: 10.1007/s12640-020-00252-7

Figure 3:

Figure 3:

The 2000 mg/kg dose of HPβCD reduced or abolished high-frequency DPOAE. Mean (n=8) DPOAE input/output function obtained pre-treatment (shaded area: 95% confidence interval) and two days (+/−SEM) and eight weeks (+/− SEM) post treatment. Down arrow denotes approximate pre-treatment DPOAE threshold at each frequency. DPOAE input/output functions shown at f2 frequencies of (A) 4 kHz, (B) 8 kHz, (C) 16 kHz, (D) 24 kHz, (E) 30 kHz and (F) 45 kHz. (A) 4 kHz DPOAE amplitudes reduced two days post treatment; however, input/output function fully recovered by eight weeks post treatment. (B-C) 8 kHz and 16 kHz DPOAE amplitudes moderately reduced at two days post treatment; DPOAE amplitude nearly fully recovered by eight weeks post treatment. (D-E) DPOAE amplitudes greatly reduced two days post treatment and only partially recovered by eight weeks post exposure. (F) 45 kHz DPOAE abolished at two days and eight weeks post treatment.