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. Author manuscript; available in PMC: 2020 Sep 17.
Published in final edited form as: Biochem Biophys Res Commun. 2020 Jun 15;530(2):432–439. doi: 10.1016/j.bbrc.2020.05.070

Fig. 2.

Fig. 2.

Hepatic expression of CIDE family and CREBH by HNF4α in human hepatoma cells. (A) quantitative RT-PCR from total RNA of HepG2 and Huh7 cells treated with negative control of siRNA (siCont) and siRNA for HNF4A (siHNF4A). (B) quantitative RT-PCR from total RNA of HNF4α-transfected Huh7 cells. (C) quantitative RT-PCR from total RNA of HLE/tet-HNF4A cells treated without or with doxycycline (−DOX, or + DOX) for 24 h. The normalized expression in Hnf4aΔHep mice, HNF4α-transfected cells, and the cells treated with doxycycline is presented relative to that in Hnf4af/f mice, empty vector-transfected cells, and the cells treated without doxycycline, respectively. Data are mean ± S.D. *, P < 0.05; **, P < 0.005 compared to the cells treated with negative control of siRNA, the cells transfected an empty, or the cells treated without doxycycline. a, P < 0.05; b, P < 0.01 compared to the cells transfected an empty. *, P < 0.05; #, P < 0.01 compared to the cells transfected CREBH(N) vector. ND; not detected.