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. Author manuscript; available in PMC: 2021 Oct 1.
Published in final edited form as: Free Radic Biol Med. 2020 Jul 23;158:162–170. doi: 10.1016/j.freeradbiomed.2020.06.006

Figure 4. Deletion of p53 increased PRDX 3 content.

Figure 4.

The contents of PRDX 3 and PRDX 5 were measured using immunoblotting. In mitochondria isolated from non-ischemic control mice, PRDX 3 content was increased in p53 deletion mitochondria compared to wild type mice in the baseline condition (Panel A). The PRDX3 content remained greater in p53 deletion mice following ischemia-reperfusion compared to wild type (Panel B). However, PRDX 5 content was not altered in p53 deletion mice compared to wild type mice (Panel C). Subunit 4 of cytochrome oxidase was used as a loading control. (Mean ± SD. *p <0.05 vs. wildtype; n = 4 in each group)