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. 2020 Sep 4;18:140. doi: 10.1186/s12964-020-00648-0

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — High CXCL10 plasma levels is associated with AATD mediated-liver fibrosis in AATD individuals. a Dot plot graph illustrating differences in the serum CXCL10 levels of ZZ individuals compared to MM healthy controls, *P = 0.0131. b The correlation between serum CXCL10 levels and α-SMA immunoreactivity on the liver biopsy from ZZ individuals. c The correlation between EV associated INF- γ, IL1- β and TNF- α levels of and liver fibrosis score of ZZ individuals. d Mild and severe perisinusoidal α-SMA staining on the liver biopsy from ZZ individuals with low and high CXCL10 plasma levels respectively