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. 2020 Sep 7;20:416. doi: 10.1186/s12870-020-02613-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — The RTM-GWAS results of BW. a Manhattan (left) and quantile–quantile plots (right) of BW. b Distribution of allele effect of the significant SNPLDB loci for BW. The number 1 indicates the significant SNPLDB locus LDB_25_61293136. c The BW differences of the stable SNPLDB locus LDB_25_61293136 among different alleles. Allele1–3 represent AA, AC and CC, respectively. * and ** indicate respectively 5% and 1% significance level