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. 2020 Sep 14;16:334. doi: 10.1186/s12917-020-02549-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Diverse antigenic reactivity of mAbs to PCV2 genotypes. 200 TCID₅₀ of QIA215, QIA418, and two PCV2d isolates (QIA169 and QIA244) were infected to PK15 cells, and the infected cells were reacted with mAb-1 (A), mAb-2 (B), mAb-3 (C), mAb-4 (D), mAb-5 (E), and mAb-6 (F). The average of fluorescent intensity per 1 × 10⁴ cellular nuclei was measured by ArrayScan VTI HCS. Different superscript letters indicate significant differences (p < 0.05)