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. 2020 Sep 14;15:53. doi: 10.1186/s13024-020-00393-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Non-convulsive epileptiform activity. Intracranial EEG recordings were obtained in the resting state from 8 to 12-month-old genetically modified mice (+) and WT controls (−). a Traces depict typical epileptiform spikes. b Spike frequencies measured during a 12-h period. c Representative SWDs. d, e Frequency (d) and length (e) of SWDs measured during a 12-h period. n = 10–12 mice per group. f–h Power spectral density analysis (n = 5 mice per group). Two waveforms were analyzed per genetically modified mouse. Note the spectral peak at 12 Hz. **P < 0.01, ***P < 0.001, ****P < 0.0001 vs. WT from same line or as indicated by brackets, based on one-way ANOVA followed by Holm-Sidak test. Values are means ± SEM