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. 2020 Sep 14;20:878. doi: 10.1186/s12885-020-07343-w

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — ANGPTL4 overexpression inhibits cell migration, invasion and adhesion in invasive breast cancer cell lines. a The expression of ANGPTL4 in luminal BC cell lines such as SKRB3 and MDA-MB-453 is higher than that in basal-like BC cell lines such as BT549 and MDA-MB-231. b The efficiency of transfection was confirmed by Western blots. Stable ANGPTL4-overexpressing cells (abbreviated BT549 and 231 ANGPTL4 OE) exhibited weaker wound healing (c), Matrigel invasion (d) and attachment abilities than the negative control (abbreviated NC) and empty vector cells (Graghpad Prism statistical software package, version 5.01, The Student’s t-test was used, *p < 0.05)