Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Sep 7;12:8093–8103. doi: 10.2147/CMAR.S264674

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2020 Qu et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

PMC Copyright notice

Effects of miR-646 inhibitor and circFLNA silencing on GC progression. HGC27 and AGS cells were transfected with sh-NC, sh-circFLNA#3, sh-circFLNA#3 + in-miR-NC or sh-circFLNA#3 + in-miR-646. (A) QRT-PCR was used to detect the expression of miR-646. CCK8 assay (B-C), transwell assay (D-E) and flow cytometry (F) were used to determine the proliferation, migration, invasion and apoptosis, respectively. The lactate production (G), glucose consumption (H) and ATP level (I) of cells were measured by Lactate Assay Kit, Glucose Assay Kit and ATP Assay Kit, respectively. (J) Glucose Uptake Assay Kit was employed to evaluate the glucose uptake of cells. *P < 0.05, **P < 0.01, ***P < 0.001.