Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Sep 8;12:8183–8195. doi: 10.2147/CMAR.S263586

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2020 Liu et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

PMC Copyright notice

CircTTBK2 level was increased in glioma tissues and cells. (A) The expression level of circTTBK2 in glioma tissues and normal tissues was detected through qRT-PCR assay. (B) Fold change of circTTBK2 expression level was presented as log2 (tumor/normal). (C) The overall survival of glioma patients with high/low circTTBK2 expression level was analyzed by Kaplan-Meier survival analysis. (D) The expression level of circTTBK2 in T98G, LN229 and NHA cells was determined by qRT-PCR assay. (E) After T98G cells were treated with Act D at indicated time points, the abundance of circTTBK2 and TTBK2 was examined via qRT-PCR assay. (F) The expression levels of circTTBK2 and TTBK2 mRNA in T98G cells treated with or without RNase R were examined by qRT-PCR assay. *P<0.05.