Table 2.
Details in patients with central venous obstructions and anatomic anomalies
| Recurrent VTE N = 32 |
No recurrent VTE N = 24 |
Total N = 56 |
||
|---|---|---|---|---|
| 6 (18.8) | 5 (20.8) | 11 (19.6) | ||
| Anatomic anomalies | ||||
| #1 | Duplication of the VP, fibrosis of the VF | #1 | Aneurysm VP | |
| #2 | Duplication of the VF | #2 | Duplication and fibrosis of the VF | |
| Central venous obstructions | ||||
| #3 | Extraluminal compression: CIV and EIV | #3 | Extraluminal compression: ICVir and CIV | |
| #4 | Extraluminal compression: CIVa | #4 | Extraluminal compression: ICVir and CIV | |
| #5 | Extraluminal compression: ICVir and CIVb | #5 | Extraluminal compression: CIVc | |
| #6 | Extraluminal compression: CIVc | |||
Data are n (%)
ICVir Inferior caval vein, infra renal, CIV Common iliac vein, EIV External iliac vein, FV Femoral vein, PV Popliteal vein, VTE Venous thrombo-embolism
None of the variables mentioned in this table showed statistical significant difference between groups
Venous obstruction is defined as either extraluminal compression (e.g. due to May-Thurner Syndrome, adjacent anatomical structures, pelvic tumour) or the presence of anatomical anomalies (e.g. agenesis, hypoplasia, aneurysms, anatomical variances, and duplications) that might negatively influence the central venous flow
a Extraluminal compression caused by spondylosis
b Extraluminal compression caused by the left iliac artery
c Extraluminal compression caused by May Thurner Syndrome (compression by the right iliac artery)