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. 2020 Sep 16;11:4678. doi: 10.1038/s41467-020-18276-0

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© This is a U.S. government work and not under copyright protection in the U.S.; foreign copyright protection may apply 2020

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Fig. 8 — It demonstrates similar wound-activated signature of genes involved in differentiation, cytokines, and intermediate filaments. Inhibition of FOXM1, STAT3, and TNFα regulators results in lack of immune-cell activation, proliferation and survival in the DFU environment contributing to deregulated inflammatory response and inhibition of wound healing.