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. 2020 Sep 17;11(9):766. doi: 10.1038/s41419-020-02938-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Differentially expressed Nrf2 was determined using high-throughput sequencing (mean ± SD, **p < 0.01). b The relationship between the expression of Nrf2 transcripts in pancreatic cancer patients. c A K-M analysis indicated that high expression of Nrf2 was associated with pancreatic cancer patients (p = 0.045). d–g After PC cells were treated with or without anlotinib or siNrf2, the cells were stained with PI and Annexin-V-FITC and detected by flow cytometry (mean ± SD, *p < 0.05; **p < 0.01; ***p < 0.001). h After PC cells were treated with or without anlotinib or siNrf2, several markers reflecting the apoptosis level were detected by western blotting. i Several unfolded protein response markers were detected by western blotting.