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. 2020 Sep 18;16:345. doi: 10.1186/s12917-020-02568-0

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — The mechanism of inhibition of PCV2-induced mitochondrial apoptosis by Cepharanthine or Curcumin. After cells were infected with 10^4.4 TCID₅₀ of PCV2 for 2 h, cells were treated with Cepharanthine, Curcumin or Ribavirin for 48 h. a-h The expression of the apoptin were analyzed by Western blot and grayscale analysis. h, i The results of flow cytometry showed that ROS values were significantly decreased in the treated group with Cepharanthine at 0.003 and Curcumin at 0.02, 0.01 and 0.005 mg/mL Compared to the PCV2-infected group (P < 0.05)