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Influence of exogenous GRK2 or GRK3 overexpression on β-arrestin2 recruitment. HEK293T cells stably co-expressing ELucN-βarr2 and the DxR-ELucC were transiently transfected with GRK2/GRK3 or empty vector (A,B). Data represent means ± SEM from three independent experiments, each performed in triplicate or quadruplicate.
