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. 2020 Aug 13;40(10):2454–2467. doi: 10.1161/ATVBAHA.120.315028

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© 2020 The Authors.

Arteriosclerosis, Thrombosis, and Vascular Biology is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDerivs License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 1. — Atlas of the hind limb skeletal muscles of C57BL/6J mice subjected to femoral artery excision. A–C, Maps depicting the skeletal muscles in the proximal (A), mid- (B), and distal mouse hind limb (C). Adjacent to each map is a corresponding hematoxylin and eosin–stained full cross section of the hind limb, 10 d after femoral artery excision. Varying intensities of muscle eosinophilia can be seen; less intense staining is present in territories of ischemic injury. D, High-magnification images, corresponding to the outlined zones within the proximal and distal hind limb, depicting normal (left) and injured (right) regions of the same muscle sections. The uninjured regions have peripheral myofiber nuclei (arrowheads). The injured/regenerating regions have pale myofibers with centralized nuclei (arrowheads).