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. 2020 Aug 13;40(10):2376–2390. doi: 10.1161/ATVBAHA.120.314284

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© 2020 The Authors.

Arteriosclerosis, Thrombosis, and Vascular Biology is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDerivs License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 5. — Effects of CORM-A1 on glycolysis and pentose phosphate pathway (PPP) metabolites in platelets. Human washed platelets (WP) suspended in PBS containing glucose (1 g/L) and glutamine (2 mmol/L) were untreated (8 min; C), treated with CORM-A1 (300 μmol/L; 8 min; A1), treated with thrombin (0.1 U/mL; T) or treated with CORM-A1 and thrombin (2+6 min; A1/T). Data show amounts of metabolic by-products in glycolysis and PPP. Data are presented as means±SD (nmoL/10⁹ platelets) of 4 independent experiments. DHAP indicates dihydroxyacetone phosphate; F-1,6-BP, fructose-1,6-bis-phosphate; GA3P, glyceraldehyde 3-phosphate; PEP, phosphoenolpyruvate; and TCA, tricarboxylic acid cycle. *P<0.05 vs control.