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. 2020 Sep 21;10:15407. doi: 10.1038/s41598-020-72325-8

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Luteolin modulated SERCA2a via up-regulating Sp1 to inhibit apoptotic proteins expression during myocardial I/R injury. (A–D) Expression of Bcl-2 and Bax proteins in each treatment group. (E, F) Expression of Cleaved caspase 3 and Caspase 3 proteins in each treatment group. Data are represented as mean ± SEM (n = 5). The full-length blots are presented in Supplemental Fig. 4. ^***P < 0.001 versus sham; ^##P < 0.01, ^###P < 0.001 versus I/R; ^&&&P < 0.001 versus Lut + I/R; ^{^}P < 0.05 versus Ad-Sp1 shRNA + I/R.