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. 2020 Sep 22;11(9):786. doi: 10.1038/s41419-020-03000-z

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — C57BL/6J mice were subcutaneously implanted with untreated GL261s, then the allografts were irradiated and treated with Bip inhibitor. After removal of primary tumors, the mice were intracranially implanted with untreated live GL261s cells. a The schedules of therapeutic regimen were indicated. b Survival curves of mice was analyzed under indicated therapeutic regimen. c Tumor size were measured after live GL261s rechallenge. d Bip gene expression has a negative correlation with CD8⁺ T cell infiltration. e Bip gene expression and GBM patients’ survival showed a negative correlation. *P < 0.05, **P < 0.01.