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. 2020 Sep 23;20:291. doi: 10.1186/s12906-020-03088-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — QCHS inhibited the expression of surface molecules and cytokines of DCs in TNBS-induced experimental colitis mice. a The frequency of MHC-II⁺CD86⁺ DCs in the MLN was measured by flow cytometry. QCHS (3, 6 and 12 g/kg) and SASP downregulated the percentage of mature DCs. b IL-12 was analyzed by ELISA. The concentration of IL-12 was reduced in a QCHS dose-dependent pattern. The data are expressed as mean ± SD, n = 6 per group. *P < 0.05 indicates a significant difference vs the control group. ^#P < 0.05 indicates a significant difference vs the TNBS group