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. 2020 Feb 23;21(5):452–462. doi: 10.1080/15384047.2020.1727700

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© 2020 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited, and is not altered, transformed, or built upon in any way.

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Figure 2. — Interference of CCAT1 suppresses the resistance of PTX in PTX-resistant PCa cells.

(a) The expression of CCAT1 was measured in PC3-TXR and DU-145-TXR cells transfected with siCCAT1 or scramble by qRT-PCR. The survival rate, IC50 of PTX (b), apoptosis (c), viability (d), C-caspase 3 expression (e) and protein levels of BCL-2, BCL-XL and MCL-1 (f) were detected in PC3-TXR and DU-145-TXR cells transfected with siCCAT1 or scramble after treatment of PTX for 24 h by MTT assay, flow cytometry and western blot assays, respectively. *P < .05.