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. 2020 Sep 25;13:128. doi: 10.1186/s13045-020-00941-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Clinical implication of AML progenitor cells from diagnosis to relapse. a, c, e UMAP analysis of normal and AML individuals before and after treatment regimen. The AML individuals are in remission (P-extra 1, a), relapse (P20, c), and partial remission (P04, e). The normal sample is N02. Colors represent clusters in left panels, and individuals in the right panels. b, d, f Relative proportion analysis of clusters in normal and AML individuals. Cell types are labeled in different colors. g–i Circos plots showing the correlation of clusters. The similar clusters are connected by lines. j–l Heatmap of differentially expressed marker genes. Each row represents marker genes of each cell type. Each column represents cells in different status. Red and yellow represents high expression level while blue represents low levels