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. 2020 Sep 24;10:15583. doi: 10.1038/s41598-020-72638-8

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Cpt1a-mutated mice have changes in their gut microbiota compared to WT mice. (a) Shannon and Simpson α-diversity measures in male Cpt1a-mutated (n = 6) and WT mice (n = 6). (b) The five most abundant bacterial communities at phyla level in male Cpt1a-mutated (n = 6) and WT mice (n = 6). (c) The ten most abundant bacterial communities at family level in male Cpt1a-mutated (n = 6) and WT mice (n = 6). (d) The 20 most abundant bacterial communities at genus level in male Cpt1a-mutated (n = 6) and WT mice (n = 6). Heat maps illustrate the relative abundance of the bacterial communities. Abbreviations: Cpt1a, carnitine palmitoyl transferase 1a with a p479l mutation; WT, wild type.