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. 2020 Sep 26;9:25. doi: 10.1186/s40164-020-00182-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — miR‑100 targeted the regulation of mTOR expression. a The binding site located between miR-100 and the mTOR-3′ of mTOR mRNA. b Prediction of the miRNA targeting the mTOR gene using bioinformatics software. c The dual‑luciferase assay showed that the mTOR-3′UTR-wt + miR-100 group significantly decreased the relative luciferase activity of HEK293T cells compared to the mTOR-3′ UTR-mut + miR-100 group (p < 0.0001).