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The possible mechanism of m6A methylation in bladder cancer. METTL3 promoted the AFF4/NF‐κB/MYC signalling network, the translation of CDCP1 and ITGA6, and inhibited the expression of SETD7 and KLF4 in an m6A‐dependent manner to further promote the carcinogenesis in bladder cancer. Different readers functioned differently and played crucial roles in bladder cancer (YTHDF1/2/3)
