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. Author manuscript; available in PMC: 2020 Sep 29.
Published in final edited form as: J Cancer Immunol (Wilmington). 2020;2(3):116–127. doi: 10.33696/cancerimmunol.2.024

Figure 1.

Figure 1.

DNA replication slippages at microsatellites generates intermediate mutations that become permanent mutations in progeny cells if not repaired by MMR, leading to MSI (A); Frameshifts in coding microsatellites inactivates gene expression and generates neo-antigenic peptides (B).