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. 2020 Sep 29;27:15. doi: 10.1186/s40709-020-00125-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — The T-cell, B-cell and plasma cell infiltration as determined by immunostaining of post‐ischemic kidney tissue with CD3, CD20 and CD138 antibodies (mouse monoclonal antibody, Ventana company, Arizona, USA). Ten resuscitated and 14 non-resuscitated stained specimens were viewed and counted for positively stained cells. Infiltration at non-resuscitated specimens (blue bars) at 24 h post‐ischemia was increased compared to the infiltration at resuscitated specimens (red bars). Both in non-resuscitated and resuscitated specimens, the infiltration of T-cell was greater than the infiltration of B-cell while there is no strong evidence of plasma cell infiltration