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. 2020 Oct 1;7:159. doi: 10.1038/s41438-020-00375-0

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Proportion of site missingness across all markers by subpopulation. b Proportion of site heterozygousity across all markers by subpopulation. c Pooled minor (second most common) allele frequency by subpopulation. d Unique polymorphic SNPs by subpopulation. e SNP density by chromosome (chr1-chr9) and genomic position in 1 Mbp windows (x-axis). f Linkage disequilibrium decay plotted as pairwise r² against log₁₀(bp) with subpopulation data fitted with cubic smoothing spline (spar = 0.5)