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. 2020 Oct 1;20:475. doi: 10.1186/s12935-020-01573-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Cell migration in colorectal cancer was inhibited by miR-138-5p. a The best mimic and inhibitor sequences were screened out. b Effectiveness of lentiviral transfection in DLD1 and HCT116 cell lines were verified. p < 0.001. c Cell migration was detected by scratch experiment. d In DLD1 cell lines, LV-inhibitor group exhibited higher percentage of cell migration than LV-INC group (p < 0.001). e, f In HCT116 cell lines, the LV-mimics group was lower in the percentage of cell number compared with LV-mNC group (p < 0.001). In DLD1 cell lines, the LV-INC group was lower in the percentage of cell number compared with LV-inhibitors group (p < 0.001)