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HSV-1 evades DDR, ER stress, autophagy, stress granules (SGs), and apoptosis. HSV-1 infection could elicit some other immune responses like SGs, ER stress, apoptosis, DDR, and autophagy that contribute to restricting viral infection. However, these immune responses are also manipulated by viral proteins. Solid lines indicate confirmed interactions between adaptors and HSV-1 proteins. P, phosphate; U, ubiquitin.
