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. Author manuscript; available in PMC: 2021 Nov 1.
Published in final edited form as: Angiogenesis. 2020 May 18;23(4):599–610. doi: 10.1007/s10456-020-09728-8

Figure 4.

Figure 4.

TEX directly and indirectly stimulate A2BR-dependent endothelial cell growth. (A) Proliferation of HUVECs in response to the treatment with CADO (1 μM) in the presence or absence of an A2BR antagonist (0.3 μM). (B) Proliferation of HUVECs in response to the treatment with TEX (10μg exosomal protein) in the presence or absence of an A2BR antagonist (0.3 μM). (C) Proliferation of HUVECs in response to the treatment with conditioned medium from Mϕ macrophages, macrophages treated with TEX and macrophages treated with TEX in the presence of an A2BR antagonist. Values are presented as fold of CTRL. All values in this figure represent means ± SEM. *p < 0.05 vs. CTRL; **p < 0.01 vs. CTRL; #p < 0.05 vs. CADO or TEX.