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. 2020 Oct 5;11:4995. doi: 10.1038/s41467-020-18819-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Disease-specific survival based on TP53m and CTNNB1m status; labelled hazard ratio represents comparison of the TP53wt/CTNNB1m group with the TP53m group. HR for TP53wt/CTNNB1wt vs. TP53m = 0.37, 95% CI 0.18–78; HR for TP53wt/CTNNB1m vs. TP53wt/CTNNB1wt = 0.31, 95% CI 0.11–0.88. b Summary of the PRISTINE algorithm for molecular subtyping in endometrioid ovarian carcinoma. m mutant, wt wild-type, RD residual disease, WES whole exome sequencing, DSS disease-specific survival.