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. 2020 Oct 2;13:9821–9837. doi: 10.2147/OTT.S265364

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© 2020 Soleymani Fard et al.

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Schematic model of the possible complex network in which AR crosstalks with apoptosis, cell cycle and EMT-related pathways to promote growth, proliferation and metastasis of gastric cancer. (A) Metastatic gastric cancer cells. (B) Inhibition of invasion by ENZ in gastric cancer cells. AR inhibition using ENZ suppressed metastatic properties of the cells through modulation of EMT and survival related-gene expressions.