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. 2020 Oct 6;6:97. doi: 10.1038/s41420-020-00333-8

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Representative images of NF200 (red) and MBP (green) immunohistochemical staining on day 14 after injury in spinal cord lesion areas. Two columns (1, and 2 respectively represent SCI group and PN group) are the enlarged images of the area around the damage boundary. Lesion core (LC) and lesion adjacent (LA) of the cavity is distinguished by the dashed lines. All cell nuclei were counterstained with DAPI (blue). Scale bar, 500 μm (upper) and 200 μm (below). b, c Semiquantification of MBP and NF200 intensity decrease in a. d Representative images of NF200 (red) and GAP43 (green) immunohistochemical staining on day 14 after injury in the two groups. All cell nuclei were counterstained with DAPI (blue). Scale bar, 200 μm. e Semiquantification of GAP43 intensity increase in d. All data are presented as means ± SEM (n = 5 mice per group). *P < 0.05.