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. 2020 Sep 23;11:548943. doi: 10.3389/fphys.2020.548943

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Copyright © 2020 De Munck, De Moudt, Roth, De Meyer, Martinet and Fransen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Ang-II treatment causes cardiac hypertrophy in Atg7^F/F SM22α-Cre⁺ mice. Ejection fraction (A), fractional shortening (B), stroke volume (C), left ventricular (LV) mass (D), LV mass corrected for body weight (BW) (E), and heart weight (HW) corrected for BW (F) of Atg7^+/+ SM22α-Cre⁺ (+/+) and Atg7^F/F SM22α-Cre⁺ (F/F) control and AngII-treated mice. (n = 6–7) Two-way ANOVA with Sidak post hoc test. p < 0.05 for genotype factor for left ventricular mass, stroke volume and LVmass/BW and for angiotensin factor with LVmass/BW. p < 0.01 for angiotensin II factor with stroke volume and for genotype factor with HW/BW. p < 0.001 for angiotensin II factor with HW/BW. *p < 0.05, **p < 0.01.