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. 2020 Oct 6;10:16638. doi: 10.1038/s41598-020-73636-6

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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BPA treatment originated an increase in the protein expression of tenascin-C and β-catenin. Data are the means ± SEM of three different experiments, each performed in duplicate. *p < 0.05 using ANOVA test. Due to BPA modified the housekeeping proteins (actin, tubulin, etc.) we used coomassie-blue staining to normalized total protein amount. Discontinue line indicates the absent lane of 1 nM BPA treatment. Full-length blots/gels are presented in Supplementary Figs. 9–10.