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. 2020 Oct 6;13:505. doi: 10.1186/s13071-020-04377-8

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Histopathological changes in the intestine during the early phase of infection by T. spiralis. Hematoxylin and eosin staining (a, b, e) and Hematoxylin/Alcyan Blue-PAS staining (c, d). Sections from non-infected (a) and infected rat small intestine showing villi atrophy (b), goblet cell hyperplasia and mucus hypersecretion (d) surrounding the parasite in the intestinal lumen (c, white arrow) and intestinal mucosae damage caused by the AW (d, black arrow). a–d original magnification 10×; e and f original magnification 20×; a is normal rat intestine while b–f are from rat intestine on 6 dpi. Scale-bars: a, b, e, f, 100 µm; c, d, 50 µm