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. 2020 Oct 6;20:482. doi: 10.1186/s12935-020-01585-1

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — −18q and sensitivity to Cabozantinib and Sorafenib. Reproduced from TCGA, shown were a overall survival, b progression-free survival and c clinicopathological parameters of patient with and without +20q; Gene enrichment and functional annotation analysis in TCGA cases with or without +20q using the d NET-GE and e GSEA platforms with genes of interest shown in heatmap; f Reproduced from TCGA dataset, shown was correlation between copy number against corresponding mRNA expression of the gene of interest; g Reproduced from TCGA and Human Protein Atlas dataset, shown was overall survival of patients with higher and lower ARHGAP28 expression with automatically designated cutoff value