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. 2020 Oct 9;3:564. doi: 10.1038/s42003-020-01278-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Sequence identity of influenza A proteins between different H1N1, H2N2, and H3N2 strains. Sequence identity is expressed as percent amino acid overlap with A/California/07/2009 (H1N1). As H1N1 does not express PB1-F2, all PB1-F2 sequences have been excluded from analysis. b Influenza epitopes presented by human MHC class-I were mapped against the protein sequence of H1N1 and A/Singapore/1/57 (H2N2). Conserved epitopes (dark blue) are present in both H1N1 and H2N2, while H1N1-specific epitopes (light blue) are only present in H1N1.