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. 2020 Oct 12;6:102. doi: 10.1038/s41420-020-00338-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A ACEA restored cell viability in HT22 cells exposed to OGD/R. B ACEA decreased lactate dehydrogenase (LDH) release from HT22 cells. C The ACEA-induced effects on cell apoptosis in OGD/R-induced HT22 cells were measured by flow cytometry. D ACEA inhibited cell apoptosis in OGD/R-induced HT22 cells. E ACEA reduced cleaved caspase-3 expression in OGD/R-induced HT22 cells. *p < 0.05; NS no significance.