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. 2020 Oct 12;6:102. doi: 10.1038/s41420-020-00338-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — ATTC staining of rat brain slices. B The CB1 receptor antagonist AM251 reversed the ACEA-induced effect on brain infarct volume in rats. C AM251 reversed the ACEA-induced amelioration of neurological function in rats. D TUNEL staining of brain tissue. E AM251 reversed the ACEA-induced inhibition of cell apoptosis. Bar = 40 µm; *p < 0.05; NS no significance.