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. 2020 Oct 13;40(21):e00294-20. doi: 10.1128/MCB.00294-20

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FIG 5 — Collectively deleting Btn2, Cur1, and Hsp42 stabilizes mutant [URE3] prions. Wild-type (wt) and btn2Δ cur1Δ hsp42Δ triple mutant (bΔ,cΔ,42Δ) cells propagating [URE3] composed of the indicated Ure2 proteins were grown as patches of cells on −ade plates to select for [URE3] and then were replica plated onto similar plates (−ade, left) and onto 1/2YPD (right). [URE3]^S158F and [URE3]^T249A are stable in the cells lacking Btn2, Cur1, and Hsp42, as indicated by the white color of cells on 1/2YPD.