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. 2020 Oct 14;20:996. doi: 10.1186/s12885-020-07509-6

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — The influence of exosomes from YB-1-upregulated GC cells on the proliferation, apoptosis, migration and invasion of HUVECs. a MTT assays revealed that SGC-7901-OE-Exo promoted the proliferation of HUVECs. b Apoptosis assays using flow cytometry and (c) the corresponding quantitative analysis demonstrated that SGC-7901-OE-Exo reduced the apoptosis of HUVECs. d Scratch wound healing assays for HUVECs in different conditions. e The level of cell migration into the wound scratch was quantified as the wound healing area and compared to that of control group at 6 and 12 h. f The transwell pictures and (g, h) the quantitative data revealed significantly increased migration and invasion of HUVECs treated by SGC-7901-OE-Exo than the control group. (*, P < 0.05; **, P < 0.01)